GABARAPs interact directly with NUFIP2 and G3BP1 whereas Atg8ylation is needed for their recruitment to damaged lysosomes. At the lysosome, NUFIP2 contributes to MTOR inactivation together with LGALS8 ...

Mishima, Y., and Schaub, jun., F. F., Proc. Twelfth Intern. Cong. Dermatol. Excerpta Med. Intern. Cong., Series 55, 1588 (1963).

This process involves the formation of autophagosomes that ultimately fuse with lysosomes to degrade cellular components ... and mammalian target of rapamycin (mTOR), effectively promoting or ...

Oxidative Stress: Neuropathy, Excitability, and Neurodegeneration.

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FUNDC1 plays an important role in mitochondrial events, participating in mitochondrial fission, maintaining the homeostasis of iron and proteins in mitochondrial matrix, and mediating crosstalk ...

Macroautophagy (hereafter autophagy) is an intracellular degradation system delivering cytoplasmic components to the lysosome via autophagosomes (Mizushima and Komatsu, 2011). Autophagy contributes to ...

The main idea tested in this work is that host galectin-9 inhibits Mycobacterium tuberculosis (Mtb) growth by recognizing the Mtb cell wall component arabinogalactan (AG) and, as a result, disrupting ...

Retraction Note: M2 macrophage-derived extracellular vesicles promote gastric cancer progression via a microRNA-130b-3p/MLL3/GRHL2 signaling cascade. Retraction Note ...